Adamax

N-acetyl adamantyl Semax analogueCognition/Mood

Evidence grade

DResearch/compounded/adjunct (no established FDA indication)

Cardiovascular · 0–5 · separate axis

PurposeNootropic / Neuro

MechanismA Semax (ACTH 4-10) analogue carrying an N-terminal acetyl group and a C-terminal adamantane, intended to resist proteolysis and improve cellular uptake.

EffectsProposed to act on BDNF and TrkB signalling by analogy with Semax, which raises BDNF and NGF-related signalling in animal models.

DosingNo human dose established.

Bottom lineZero published studies on Adamax itself. Everything claimed for it is borrowed from Semax.

Batches on filenone

No tested batches on file.

Read with

The library records no pairings for this entry.

Cautions & interactions

A literature search returns no dedicated peer-reviewed human trials and effectively no published preclinical work on the modified molecule. Whether the adamantane modification improves on Semax in humans is simply unknown. Sold for laboratory research only.

No interaction data exists.

CV 1 of 5 — No cardiovascular data.